Publications

Found 14 results
Filters: Keyword is Calcium-Calmodulin-Dependent Protein Kinase Type 2  [Clear All Filters]
2012
Flynn R, Labrie-Dion É, Bernier N, Colicos MA, De Koninck P, Zamponi GW.  2012.  Activity-dependent subcellular cotrafficking of the small GTPase Rem2 and Ca2+/CaM-dependent protein kinase IIα.. PLoS One. 7(7):e41185.
Fatimy REl, Tremblay S, Dury AY, Solomon S, De Koninck P, Schrader JW, Khandjian EW.  2012.  Fragile X mental retardation protein interacts with the RNA-binding protein Caprin1 in neuronal RiboNucleoProtein complexes [corrected].. PLoS One. 7(6):e39338.
El Gaamouch F, Buisson A, Moustié O, Lemieux M, Labrecque S, Bontempi B, De Koninck P, Nicole O.  2012.  Interaction between αCaMKII and GluN2B controls ERK-dependent plasticity.. J Neurosci. 32(31):10767-79.
Lemieux M, Labrecque S, Tardif C, Labrie-Dion É, Lebel E, De Koninck P.  2012.  Translocation of CaMKII to dendritic microtubules supports the plasticity of local synapses.. J Cell Biol. 198(6):1055-73.
2010
Pi HJae, Otmakhov N, Lemelin D, De Koninck P, Lisman J.  2010.  Autonomous CaMKII can promote either long-term potentiation or long-term depression, depending on the state of T305/T306 phosphorylation.. J Neurosci. 30(26):8704-9.
Pi HJae, Otmakhov N, El Gaamouch F, Lemelin D, De Koninck P, Lisman J.  2010.  CaMKII control of spine size and synaptic strength: role of phosphorylation states and nonenzymatic action.. Proc Natl Acad Sci U S A. 107(32):14437-42.
Opazo P, Labrecque S, Tigaret CM, Frouin A, Wiseman PW, De Koninck P, Choquet D.  2010.  CaMKII triggers the diffusional trapping of surface AMPARs through phosphorylation of stargazin.. Neuron. 67(2):239-52.
2007
Taylor DM, De Koninck P, Minotti S, Durham HD.  2007.  Manipulation of protein kinases reveals different mechanisms for upregulation of heat shock proteins in motor neurons and non-neuronal cells.. Mol Cell Neurosci. 34(1):20-33.
2006
Bayer K U, Lebel E, McDonald GL, O'Leary H, Schulman H, De Koninck P.  2006.  Transition from reversible to persistent binding of CaMKII to postsynaptic sites and NR2B.. J Neurosci. 26(4):1164-74.
2005
Hudmon A, Lebel E, Roy H, Sik A, Schulman H, M Waxham N, De Koninck P.  2005.  A mechanism for Ca2+/calmodulin-dependent protein kinase II clustering at synaptic and nonsynaptic sites based on self-association.. J Neurosci. 25(30):6971-83.
2003
Dupont G, Houart G, De Koninck P.  2003.  Sensitivity of CaM kinase II to the frequency of Ca2+ oscillations: a simple model.. Cell Calcium. 34(6):485-97.
2002
Bayer K U, De Koninck P, Schulman H.  2002.  Alternative splicing modulates the frequency-dependent response of CaMKII to Ca(2+) oscillations.. EMBO J. 21(14):3590-7.
2001
Bayer K U, De Koninck P, Leonard AS, Hell JW, Schulman H.  2001.  Interaction with the NMDA receptor locks CaMKII in an active conformation.. Nature. 411(6839):801-5.
1998
De Koninck P, Schulman H.  1998.  Sensitivity of CaM kinase II to the frequency of Ca2+ oscillations.. Science. 279(5348):227-30.

Funding

Our research endeavors are made possible by the following agencies:

Canadian Institutes of Health Research - Instituts de recherche en santé du Canada Fonds de recherche du Québec – Nature et technologies (FRQNT)Fonds de la recherche en santé du Québec   Natural Sciences and Engineering Research Council of Canada (NSERC) - Conseil de recherche en sciences naturelles et en génie du Canada (CRSNG)innovation.caHuman Frontier Science ProgramCanada First Research Excellence FundSentinelle Nord